Genotype-Phenotype Studies in Brain Tumors
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8 Figures and Tables
Table 1: Simplified schedule of the most common subtypes of meningioma and glioma classifications according to the 2007 WHO classification.
Figure 2: A simplified illustration of a chromosome locus with homozygous (A) and heterozygous (B) alleles.
Table 2: Previous reported risk variants investigated in Paper III and Paper IV
Figure 3: An illustration of an association study presenting the case group (A) having a genetic variant (the grey (“alleles”) flags) over-represented compared to the control group (B).
Figure 3: Schematic structure of an LRIG protein. The extracellular part of the protein is the conserved region and there is great variation in the intracellular part. This illustration is based on a study by Holmlund et al. 2004 .
Table 3: Summarized results for detection of EGFR amplification in 19 glioblastoma patients, using the SNP array (ASCAT algorithm) and FISH analysis.
Figure 4: Illustration of normal (wild-type) IDH activity (A) versus mutant IDH activity in glioma tumorigenesis (B). This figure is redrawn and simplified from Nikiforova et al 2011 .
Figure 5: Flow chart for inclusion of patients in Paper III and Paper IV.
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